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Brain & mental-health explorer

Mental health conditions aren’t “all in your head” in the way people sometimes mean it — they involve real, understandable changes in the brain. Explore the regions below, pick a condition to see which parts light up, and switch between plain-language and clinical detail.
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Lateral brain view

Interactive brain diagram — lateral viewFrontalParietalOccipitalTemporalPrefrontalCortexAnt. CingulateInsulaThalamusHypothal.AmygdalaHippocampusN.Acc.BrainStemCerebellum

Tap any region to read about it. This is an educational illustration, not an anatomically exact scan.

Select a brain region

Tap any area on the brain, or pick one from the list below, to learn what it does and how it shows up in mental health.

All regions

Circuit view: how these regions connect

The brain works through circuits, not isolated parts. These diagrams show the key functional pathways behind each condition — arrows are connections, and color shows whether a structure runs hyperactive (warm) or hypoactive (cool) in this condition.

Depression: cortico-limbic & reward circuit

Hyperactive Hypoactive Neutral / source nucleus
Depression: cortico-limbic & reward circuitdlPFC ↔ sgACC (BA25): Top-down regulation — weakenedsgACC (BA25) ↔ Amygdala: Limbic-cortical loop — sgACC drives amygdala reactivityAmygdala ↔ Hippocampus: Emotional memory & contextual processing — impairedsgACC (BA25) ↔ PCC / precuneus: Default mode network hyperconnectivity — ruminationVentral striatum (NAcc) ↔ dlPFC: Fronto-striatal loop — reward-guided decision-makingVTA → Ventral striatum (NAcc): Mesolimbic dopamine — blunted, drives anhedoniaRaphe nuclei → Amygdala: Serotonergic projectionRaphe nuclei → Hippocampus: Serotonergic projectionLocus coeruleus → Amygdala: Noradrenergic projectionAmygdala → Hypothalamus: Drives HPA activationHypothalamus → Pituitary: CRHPituitary → Adrenal gland: ACTH → cortisolAdrenal gland → Hippocampus: Cortisol — impaired negative feedbackdlPFCexecutive controlsgACC (BA25)limbic-cortical hubPCC / precuneusdefault mode networkVentral striatum (NAcc)rewardAmygdalathreat & valenceHippocampusmemory & contextVTAdopamine sourceRaphe nucleiserotonin sourceLocus coeruleusnorepinephrine sourceHypothalamusHPA axisPituitaryHPA axisAdrenal glandcortisol

How this circuit works

Depression isn't a single "broken part" — it's a loop. A calming region at the front of the brain goes quiet just as an emotional-alarm region gets louder, the memory center loses its ability to file feelings into context, the reward pathway stops responding to things that used to feel good, and the stress-hormone system gets stuck partly "on." A network that runs when the mind is idle and turned inward (the kind of background thinking behind rumination) also gets stuck in overdrive.

Key connections

  1. dlPFC sgACC (BA25): Top-down regulation — weakened
  2. sgACC (BA25) Amygdala: Limbic-cortical loop — sgACC drives amygdala reactivity
  3. Amygdala Hippocampus: Emotional memory & contextual processing — impaired
  4. sgACC (BA25) PCC / precuneus: Default mode network hyperconnectivity — rumination
  5. Ventral striatum (NAcc) dlPFC: Fronto-striatal loop — reward-guided decision-making
  6. VTA Ventral striatum (NAcc): Mesolimbic dopamine — blunted, drives anhedonia
  7. Raphe nuclei Amygdala: Serotonergic projection
  8. Raphe nuclei Hippocampus: Serotonergic projection
  9. Locus coeruleus Amygdala: Noradrenergic projection
  10. Amygdala Hypothalamus: Drives HPA activation
  11. Hypothalamus Pituitary: CRH
  12. Pituitary Adrenal gland: ACTH → cortisol
  13. Adrenal gland Hippocampus: Cortisol — impaired negative feedback

Simplified: this collapses many DMN nodes (mPFC, PCC, precuneus, angular gyrus) into a single "PCC/precuneus" node, and shows only two monoamine nuclei explicitly (5-HT, NE) plus dopamine via the VTA — inflammatory and glutamatergic (ketamine-relevant) mechanisms aren't diagrammed.

Brain–body connection

Mental health lives in the body too. These are three of the pathways that link what happens in the brain to what you feel physically.

Stress Response (HPA Axis)

When you feel stressed, your brain sends a signal that releases the stress hormone cortisol. This affects your whole body.

  1. 🧠 Hypothalamus detects threat
  2. ⚛️ Releases CRH → Pituitary releases ACTH
  3. 💧 Adrenal glands release cortisol
  4. ❤️ Heart rate, blood pressure, digestion, immunity, sleep all affected

Anxiety & the Nervous System

When your brain's alarm center (amygdala) fires too often, it triggers your body's fight-or-flight response, causing the physical symptoms of anxiety.

  1. 🚨 Amygdala sounds the alarm
  2. ⚡ Sympathetic nervous system activates
  3. 💓 Racing heart, rapid breathing, sweating
  4. 💀 Muscle tension, stomach upset, dizziness

Depression & the Body

When brain chemicals like serotonin and dopamine are out of balance, it doesn't just affect your mood. It affects your energy, sleep, appetite, and even your ability to feel pleasure.

  1. 🔬 Serotonin & dopamine levels drop
  2. 💤 Sleep disrupted (too much or too little)
  3. 🍽️ Appetite changes (increase or decrease)
  4. 😔 Low energy, loss of motivation and pleasure

Neurotransmitter quick reference

The brain’s chemical messengers. Most mental health medications work by nudging one or more of these back into balance.

Serotonin (5-HT)

Helps regulate mood, sleep, appetite, and digestion. Often called the "feel good" chemical.

Too lowDepression, anxiety, insomnia, increased appetite, irritability
Too highSerotonin syndrome (agitation, hyperthermia, clonus, autonomic instability — medical emergency)
MedsSSRIs (fluoxetine, sertraline), SNRIs (venlafaxine, duloxetine), triptans, buspirone, trazodone
LinksDepression, Anxiety, OCD, PTSD, Eating Disorders

Dopamine (DA)

Drives motivation, reward, and pleasure. Makes you want to do things and feel good when you accomplish them.

Too lowLow motivation, anhedonia, fatigue, difficulty concentrating, Parkinson's tremor
Too highPsychosis, mania, impulsivity, compulsive behaviors, tics
MedsStimulants (methylphenidate, amphetamines), bupropion, antipsychotics (D2 blockers), L-DOPA, pramipexole
LinksADHD, Addiction, Depression, Schizophrenia, Parkinson's, Bipolar

Norepinephrine (NE)

Controls alertness, energy, and the body's "fight or flight" response. Helps you focus and respond to stress.

Too lowFatigue, poor concentration, depression, low blood pressure, brain fog
Too highAnxiety, hypervigilance, insomnia, elevated blood pressure, panic
MedsSNRIs (venlafaxine, duloxetine), atomoxetine, guanfacine (alpha-2A agonist), prazosin (alpha-1 blocker), propranolol (beta blocker)
LinksADHD, Depression, Anxiety, PTSD, Panic Disorder

GABA

The brain's main "brake pedal." It calms neural activity and helps reduce anxiety, promote sleep, and prevent seizures.

Too lowAnxiety, insomnia, seizures, muscle tension, racing thoughts
Too highExcessive sedation, respiratory depression, cognitive impairment
MedsBenzodiazepines (GABA-A PAMs), gabapentin/pregabalin (indirect), barbiturates, Z-drugs (zolpidem), valproate (increases GABA)
LinksAnxiety, Insomnia, Epilepsy, Alcohol Withdrawal, Bipolar

Glutamate

The brain's main "gas pedal." It activates neural circuits, drives learning and memory formation, and helps the brain adapt.

Too lowCognitive impairment, fatigue, insomnia, poor concentration
Too highExcitotoxicity, seizures, anxiety, neurodegenerative damage, psychosis
MedsKetamine/esketamine (NMDA antagonist), memantine, lamotrigine (reduces release), riluzole, NAC (modulates via cystine-glutamate exchanger)
LinksDepression (ketamine mechanism), Schizophrenia, OCD, Epilepsy, Alzheimer's

Endorphins

Your body's natural painkillers and pleasure chemicals. Released during exercise, laughter, and social bonding. Create feelings of well-being.

Too lowIncreased pain sensitivity, difficulty coping with stress, depression, chronic pain conditions
Too highReduced pain perception, euphoria (relevant in opioid use disorder)
MedsNaltrexone (opioid antagonist — blocks), opioid analgesics (agonists), buprenorphine (partial agonist), LDN (low-dose naltrexone — investigational for immune/pain)
LinksChronic Pain, Addiction (Opioid Use Disorder), Depression, Self-harm

A note on all of this

Brains are far more complex and individual than any diagram can show, and no one region “causes” a condition on its own. This tool is here to make the science feel a little less mysterious — not to diagnose anyone. If something here resonates, it’s worth talking through with a clinician.

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